COLORECTAL SURGERY BLOG

Diverticular disease consists of diverticulosis and diverticulitis. Individuals who have diverticulosis have small pouches in their colons which protrude outward through weak spots.. When theses pouches become inflamed or infected, the condition is referred to as diverticulitis. Approximately 50% of all Americans between the ages of 60 and 80 have diverticulosis. About 10% to 25% of individuals who have diverticulosis end up having inflamed or infected pouches of diverticulitis.

New research has exposed a possible link between frequent antibiotic use and the development of Inflammatory Bowel Disease (IBD) such as Crohn’s Disease or ulcerative colitis. Although most people who take antibiotics will not develop problems, it seems that those who do have problems generally have had to take large quantities of antibiotics.

Get the PDF version of this article HOW TO REMOVE A RECTAL FOREIGN BODY IN ONE EASY STEP (RARE IS THE PATIENT WHO EXPERIENCES THIS PROBLEM TWICE) THE END For those with a short attention span and a desire for immediate answers, here is the conclusion of this article: CONCLUSION: From a safety standpoint, the

Get the PDF version of this article COLON CLEANSING – DOES A CLEAN COLON LEAD TO A CLEAN MIND AND CLEAN BODY? THE DATA (OR LACK OF DATA) ABOUT COLON CLEANSING THE END For those with a short attention span and a desire for immediate answers, here is the conclusion of this article: CONCLUSION –

First documented in 1839, hemangiomas and vascular malformations of the GI tract are an infrequently encountered entity. Having the potential to occur anywhere along the intestinal system, the small bowel is the most frequent site with hemangiomas and malformations accounting for only 10% of all small bowel tumors.

Get the PDF version of this article FAMILIAL ADENOMATOUS POLYPOSIS: ONE GENE. MANY MANIFESTATIONS. ALPHABET SOUP: FROM HERE TO THERE AND BACK AGAIN…AGAIN Part III of this series looked at Familial Adenomatous Polyposis (FAP) and the genetics underlying the disease. Part IV will examine the ways in which this knowledge can be used to diagnose

Get the PDF version of this article FAMILIAL ADENOMATOUS POLYPOSIS: ONE GENE. MANY MANIFESTATIONS. MAKING SENSE OF THE ALPHABET SOUP THAT IS THE GENETIC CODE. Parts I and II of this series looked at the genetics, diagnosis and treatment of hereditary nonpolyposis colorectal cancer. Part III will examine familial adenomatous polyposis and the damage caused

Get the PDF version of this article Hereditary Nonpolyposis Colorectal Cancer: From Diagnosis to Treatment In this four-part series, Part 1 reviewed the basic pathologic and genetic concepts underlying hereditary nonpolyposis colorectal cancer. This second part defines the clinical challenges facing the clinician who is at the forefront of diagnostic and treatment efforts. Parts 3

Get the PDF version of this article HEREDITARY NONPOLYPOSIS COLORECTAL CANCER: GENETICS AND DIAGNOSIS THE ALPHABET SOUP OF GENETICS AND DIAGNOSIS EXPLAINED With a myriad of vexing abbreviations and obscure terminology, the genetics controlling the formation of colonic polyps and malignancies may be difficult to appreciate. Paradoxically, the physician, positioned at the beginning of the

Squamous cell cancer of the anal canal is rare and adenocarcinoma is rarer still, accounting for 10% of all anal cancers. , The three types of anal adenocarcinoma are those arising in the anorectum, those arising in anal glands, and those arising in an anorectal fistula.